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Investors were widely expecting Celgene to announce expanded European regulatory approval of Revlimid as a treatment for newly diagnosed multiple myeloma patients and as maintenance therapy following stem cell transplant. But in a surprise move, the company disclosed Thursday that it was withdrawing the Revlimid submission, and at the request of European regulators, will re-submit at a later date with more mature data.

Part of the challenge of unraveling the genetic basis for nonrandom mating lies in the number of entities that may be involved. Physiologically, postpollination nonrandom mating can occur via competition among pollen for access to ovules (male-mediated nonrandom mating). Alternatively, nonrandom mating could be due to differences in the complementarities between female tissues and individual pollen grains: pistil favoritism toward some pollen donors or handicapping of some pollen donors (female-mediated nonrandom mating). The genetics of this process have the potential to extend beyond the gametophyte genomes. Heritable differences in pollen tube growth may be encoded by the paternal sporophyte, which preloads pollen grains with nutrients for the initial stages of pollen tube growth, as well as by the male gametophyte genome (Stephenson et al., 2003). If anything, things may be more complex on the female side of the equation. Female-mediated nonrandom mating may be influenced by the obvious female sporophyte tissue that pollen tubes grow through and seeds develop in as well as the female gametophyte, which plays a role in guiding the pollen tube during the last stages of fertilization (Higashiyama et al., 2001; Higashiyama, 2002; Okuda et al., 2009). This highlights the importance of assaying female and male controls separately when dissecting the physiology and genetics of this process. 041b061a72